Monday, December 11, 2017

The Role of #Fibrin in #Parkinson’s #Disease #Serrscor #Nattokinase



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Parkinson’s disease is an irreversible nervous system disorder that primarily affects musculo-skeletal movement. While the progressive disorder can cause stiffness and slow movements, signs and symptoms vary greatly among patients.

Common symptoms include: 
Tremors
Slowed Movement
Rigid Muscles
Impaired balance
Impaired Posture 
Speech changes


Parkinson’s disease is caused by the gradual breakdown of brain cells called neurons. The loss of neurons leads to a decrease in dopamine levels, leading to abnormal brain activity. Only 5% of Parkinson’s cases occur before the age of 40, with incidence increasing with age. A majority of cases seem to be sporadic and of unknown origin – however, like most diseases, there is a significant genetic component.

Recent theories about the causes of Parkinson’s disease are based on abnormal fibrin production and deposition in the brain. Also known as the “amyloid hypothesis,” researchers have been looking at the role of abnormal protein aggregation in Parkinson’s and other neurodegenerative disorders, like Alzheimer’s disease. There seems to be a correlation between fibrin deposits and brain tissue degeneration, suggesting that the fibrin contributes to cellular death in these diseases. Similar to Alzheimer’s disease and Huntington’s disease, Parkinson’s disease involves the mis-folding of a specific protein, eventually leading to fibrin deposits.

There are several theories as to the underlying cause of Parkinson’s, from oxidative damage to environmental toxins – but the latest research seeks to understand why the dopamine-containing neurons are affected so greatly. In fact, by the time a Parkinson’s patient presents with symptoms, about 70% of the dopamine-containing neurons have already been lost. Current therapies aim to address dopamine deficiencies, but future therapies seek to address the prevention of neuron degeneration (cellular death of the brain cells).

Although systemic enzymes are implicated for the treatment of Alzheimer’s disease, their use in similar neurodegenerative diseases has yet to be studied. Specifically nattokinase, which has been shown to directly dissolve the aggregated protein found in Alzheimer’s disease, may be an option available to those suffering from Parkinson’s disease.

 Nattokinase is a natural systemic enzyme that helps to decrease fibrin levels in the blood. Nattokinase is also able to reach areas where fibrin has already accumulated – helping to restore blood flow and inhibiting cellular death due to oxidative damage. Perhaps the most promising aspect of systemic enzyme therapy is its ability to be used concurrently with other medications. The only restriction is upon the use of nattokinase, which should be discussed with a doctor prior to adding it to a regimen with prescription blood thinners. However, it should be noted that systemic enzymes do not cause adverse side effects or drug-drug/drug-nutrient interactions.


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Sunday, December 10, 2017

Serrapeptase for #Respiratory #sinus #joint and #tissue health




Supports respiratory & sinus systems




Amends mucus elasticity and viscosity without causing mucus depletion



 Supports Muscle, joint and tissue health


Serrapeptase: Enteric-Coated Serrapeptase

Serrapeptase (Peptizyme-SP) for Healthy Fibrin Metabolism

Serrapeptase a powerful systemic enzyme that promotes total body support. Being the most powerful Serrapeptase on the market, Serrapeptase is unrivaled among products of its kind because it is packed with 130K SPU per capsule (80K is also available), the strongest in the world. 

Serrapeptase – The bacterium, Serratia mercesans, produces the enzyme serrapeptase to enable the silkworm to dissolve its silken cocoon and emerge as a moth after metamorphosis. Serratia is now grown in cultures to produce serrapeptase by fermentation.



What is enteric coating, and why is it needed?
Enteric coating is a layer of protective coating for dietary supplements that is meant to allow the supplement to bypass digestion by the stomach and be absorbed in the small intestine. Systemic enzymes are proteins, so their functional structures are denatured (or destroyed) in conditions of extreme pH and temperature. Without enteric coating, enzymes would be rendered useless long before they reach the bloodstream, where their carry out their beneficial effects.


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Saturday, December 9, 2017

The Common Cause of #Parkinson’s try #Exclyzme #Excellacor




The Common Cause of Parkinson’s
By William Wong ND, PhD, Member World Sports Medicine Hall of Fame

The road to discovery is long and winding. Some of its paths lead to dead ends, some of its paths curve round and round until they lead to truth. The paths trod by those seeking the cause and then a cure for the ills that effect the brain are no different.

In the search for the root cause of Alzheimer's many degenerative changes in the brain of its victims have come to light: protein cross linking short circuiting nerve transmission, aluminum shards in DNA strands, decrease in neurotransmitters, the shrinking of the brain due to loss of its fat content (the brain is 70% cholesterol), but all of these factors are the results of the condition and not its direct cause. It is now known that moderate inflammation, not enough to be called Enchaphilitis, but enough to have been medically detected, is the root cause of the condition. 

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With Parkinson’s patients a part of the brain called the Substancia Nigra dies off. This vital part of the brain makes a substance called Dopamine. It is dopamine that connects the brain to the body. As dopamine levels decrease while the Substancia Nigra dies off, slowly the control the brain exerts over the body diminishes. So essential is dopamine that doctors can tell a “pre morbid” (just before death) condition by monitoring blood levels of dopamine. Three days after the last drop of dopamine is made by the brain the person dies! A link between sub clinical brain inflammation and Parkinson’s has just been found!


Wednesday, December 6, 2017

#Serracor #Nattokinase #Fibromyalgia


 


Fibromyalgia is a disorder of chronic, widespread pain and tenderness (see the image below). It typically presents in young or middle-aged women but can affect patients of either sex and at any age.
Tender points in fibromyalgia.  
Tender points in fibromyalgia.

Signs and symptoms

Fibromyalgia is a syndrome that consists of the following signs and symptoms
  • Persistent (≥3 mo) widespread pain (pain/tenderness on both sides of the body, above and below the waist, including the axial spine [usually the paraspinus, scapular, and trapezius muscles])
  • Stiffness
  • Fatigue; disrupted and unrefreshing sleep
  • Cognitive difficulties
  • Multiple other unexplained symptoms, anxiety and/or depression, and functional impairment of activities of daily living (ADLs)

Diagnosis

Fibromyalgia is a diagnosis of exclusion and patients must be thoroughly evaluated for the presence of other disorders that could be the cause of symptoms before a diagnosis of fibromyalgia is made. The clinical assessment may reveal objective evidence for a discrete or comorbid illness, such as the following:
  • Hypothyroidism
  • Rheumatoid arthritis
  • Systemic lupus erythematosus
  • Polymyalgia rheumatic
  • Other inflammatory or autoimmune disorders
  • Serious cardiac conditions in those with chest pain, dyspnea, and palpitations
  • Social Support Questionnaire
  • Sickness Impact Profile
  • Multidimensional Pain Inventory
See Workup for more detail.

 

Management

There is no cure for fibromyalgia, but education, lifestyle changes, and proper medications can help the individual to regain control and achieve significant improvement.
Models of pain behavior that interrelate biologic, cognitive, emotional, and behavioral variables form the basis for cognitive-behavioral and operant behavioral approaches to adult pain management. Fibromyalgia in children responds to a combination of psychotherapy, exercise, relaxation techniques, and education. Pharmacotherapy is generally not indicated in children.

Nonpharmacotherapy
  • Diet (eg, promote good nutrition, vitamin supplementation, bone health, weight loss)
  • Stress management
  • Aerobic exercise (eg, low-impact aerobics, walking, water aerobics, stationary bicycle)
  • Sleep therapy (eg, education/instruction on sleep hygiene)
  • Psychologic/behavioral therapy (eg, cognitive-behavioral, operant-behavioral)

Other agents used in fibromyalgia may include the following:
  • Vitamins and minerals
  • Malic acid and magnesium combination
  • Antioxidants
  • Amino acids
  • Herbs and supplements

Monday, December 4, 2017

#Fibromyalgia TRY #Serracor-NK for #Joint #Inflammation Serracor and #Fibromyalgia Facts




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Fibromyalgia 

Before the 70's, fibromyalgia was most commonly known as fibrositis, where “it is” implied an inflammatory component. Despite the understanding of inflammatory pathways to pain, clinical research was unable to identify the role of inflammation in fibromyalgia for many years.

Within the last decade, fibromyalgia research has once again been focusing on the possible contribution of inflammation to disease progression, and is finding some new and interesting results.

Clinical studies have produced evidence that fibromyalgia is associated with the immune system’s improper regulation of proinflammatory cytokines that circulate in the bloodstream, contributing to the dysfunction of the central nervous system and pain-related neurotransmitters. Cytokines, depending on their concentration, induce symptoms, such as fatigue, fever, sleep, pain, and muscle pain, all of which develop in fibromyalgia patients.

Some experimental pain reduction therapies have been examined and shown positive results, correlating with decreased proinflammatory cytokine levels. Anticonvulsant drugs, analgesics, opiods and anti-depressants are commonly prescribed to fibromyalgia patients, but tend to carry side effects reflective of the syndrome itself, and many of which lack evidence for effectiveness.

Limited treatment options have led to an increasing use of systemic enzyme therapy as a means to alleviate symptoms and improve quality of life. Certain proteolytic (protein digesting) enzymes have been identified to have extremely beneficial actions when applied to inflammation and pain related to this condition.
 
It has long been known that people with chronic muscle pain or fibromyalgia have more fibrin in their tissues and blood. This fibrin, while initially helpful in the early stages of healing after an injury, can become problematic if the body does not clear itself of the agent after it has done its work.

Fibromyalgia sufferers experience micro-tears in their muscles from the normal activity of daily living — each and every day. But because the average fibromyalgia patient does not achieve and stay in stage 4 delta sleep at rest, growth hormone is not produced in enough quantities to heal these tears, which leads to more fibrin buildup.

For the most part, people with fibromyalgia do not have a strong enzymatic capacity for producing enzymes that break down fibrin. This leads to a buildup of fibrin, which over time catches red blood cells in a web of restriction. This fibrin causes a restriction of blood flow. Red blood cells literally become stuck, disabling them from getting into the capillaries to oxygenate and nourish the muscles where the metabolic waste that causes pain is removed.

The body uses fibrin to help heal itself after an injury. However, if you have poor blood flow and a lack of enzyme activity, fibrin will start to accumulate. If the injured area is slow to heal, fibrin accumulation appears as clumps of scar tissue in the muscles or at a surgical site.

Ultimately, if excess fibrin is present throughout the circulatory system, blood flow is restricted to areas of the body that need it most. Over time, the body compensates for this restriction by increasing its blood pressure. People with excess fibrin suffer from chronic fatigue, slow healing, inflammation and pain, as well as elevated blood pressure.

Proteolytic enzymes taken on an empty stomach break down these proteins into their smallest elements. The enzymes pass through the stomach and intestinal lining, and enter the bloodstream where they begin the process of breaking down the buildup in the muscles, connective tissue and blood. These enzymes bring nutrition and oxygen-rich blood that can remove the metabolic waste produced by inflammation and excess fibrin.

Serrapeptase has been proven to be the strongest of the proteolytic enzymes, inducing anti-inflammatory, fibrinolytic and anti-edemic (prevents swelling and fluid retention) activity in a number of tissues.
 

Using enzymes to clear your body of fibrin takes time. It takes years to develop webs of fibrin in your tissues — so be patient, log your usage and, over time, notice how much less pain and how much more flexibility you have.

Serrapeptase has demonstrated anti-inflammatory and fibrinolytic activity, and acts rapidly on localized inflammation with no reports of adverse effects.


Bromelain, a proteolytic enzyme extracted from pineapple, has also been found to be effective in reducing inflammation by blocking cytokine production and activity



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Wednesday, November 29, 2017

Digestive Enzymes & Probiotics #Floracor & #Digest-GI

 
Digestive enzymes improve the function of your digestive system and make you feel better after eating a meal. The majority of digestive support supplements contain a blend of enzymes, but with so many ingredients on every label, it can be hard to choose the right supplement for your needs.


Below are three highly effective digestive enzymes to consider when choosing a supplement.

1. Papaya
Papayas are a rich source of valuable proteolytic enzymes, such as papain, chymopapain, caricain and glycyl endopeptidase, that can greatly aid in the digestive process. The enzyme papain is one of the most effective at breaking down meat and other proteins, comparable to the enzyme pepsin that we produce in our pancreas.




In fact, papain is often considered a more effective enzyme than pepsin. Eating the papaya enzyme papain in a meal containing meat can significantly speed up its digestion. It may also help with the breakdown of other troublesome proteins, such as the gluten in wheat and the casein in milk, that are often implicated in digestive problems.


2. Bromelain
Bromelain is an enzyme found in pineapple juice and in the pineapple stem and is known to improve digestion. Used for reducing swelling and inflammation, bromelain has been shown to treat a bowel condition that includes swelling and ulcers. Often used in digestive support products, bromelain can also help improve the absorption of antibiotics. Bromelain seems to cause the body to produce substances that fight pain and inflammation.



3. Hydrochloric Acid (HCL)
Hydrochloric acid, also called HCL, is one of the many chemicals released in our stomach when we eat a meal. The role of hydrochloric acid in the stomach, along with the other gastric juices, is to break down foods and cause the release of enzymes that further aid digestion. HCl also protects the body from illness by killing pathogens commonly found in foods.



HCL supplements can aid the stomach’s acid to destroy harmful bacteria as well as relieve the symptoms of heartburn. Low stomach acid causes indigestion, gas, bloating, acid reflux, constipation, and diarrhea.


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Tuesday, November 28, 2017

#Fibromyalgia TRY #Serracor-NK for #Joint #Inflammation

Serracor and #Fibromyalgia Facts

 Buy on EBay





Fibromyalgia 

Before the 70's, fibromyalgia was most commonly known as fibrositis, where “itis” implied an inflammatory component. Despite the understanding of inflammatory pathways to pain, clinical research was unable to identify the role of inflammation in fibromyalgia for many years.

Within the last decade, fibromyalgia research has once again been focusing on the possible contribution of inflammation to disease progression, and is finding some new and interesting results.

Clinical studies have produced evidence that fibromyalgia is associated with the immune system’s improper regulation of proinflammatory cytokines that circulate in the bloodstream, contributing to the dysfunction of the central nervous system and pain-related neurotransmitters. Cytokines, depending on their concentration, induce symptoms, such as fatigue, fever, sleep, pain, and muscle pain, all of which develop in fibromyalgia patients.

These findings are uncovering new possibilities in research for fibromyalgia causation, as well as treatment options. Some experimental pain reduction therapies have been examined and shown positive results, correlating with decreased proinflammatory cytokine levels.7 Anticonvulsant drugs, analgesics, opiods and anti-depressants are commonly prescribed to fibromyalgia patients, but tend to carry side effects reflective of the syndrome itself,and many of which lack evidence for effectiveness.

Limited treatment options have led to an increasing use of systemic enzyme therapy as a means to alleviate symptoms and improve quality of life. Certain proteolytic (protein digesting) enzymes have been identified to have extremely beneficial actions when applied to inflammation and pain related to this condition.
It has long been known that people with chronic muscle pain or fibromyalgia have more fibrin in their tissues and blood. This fibrin, while initially helpful in the early stages of healing after an injury, can become problematic if the body does not clear itself of the agent after it has done its work.

Fibromyalgia sufferers experience micro-tears in their muscles from the normal activity of daily living — each and every day. But because the average fibromyalgia patient does not achieve and stay in stage 4 delta sleep at rest, growth hormone is not produced in enough quantities to heal these tears, which leads to more fibrin buildup.

For the most part, people with fibromyalgia do not have a strong enzymatic capacity for producing enzymes that break down fibrin. This leads to a buildup of fibrin, which over time catches red blood cells in a web of restriction. This fibrin causes a restriction of blood flow. Red blood cells literally become stuck, disabling them from getting into the capillaries to oxygenate and nourish the muscles where the metabolic waste that causes pain is removed.

The body uses fibrin to help heal itself after an injury. However, if you have poor blood flow and a lack of enzyme activity, fibrin will start to accumulate. If the injured area is slow to heal, fibrin accumulation appears as clumps of scar tissue in the muscles or at a surgical site.

Ultimately, if excess fibrin is present throughout the circulatory system, blood flow is restricted to areas of the body that need it most. Over time, the body compensates for this restriction by increasing its blood pressure. People with excess fibrin suffer from chronic fatigue, slow healing, inflammation and pain, as well as elevated blood pressure.

Proteolytic enzymes taken on an empty stomach break down these proteins into their smallest elements. The enzymes pass through the stomach and intestinal lining, and enter the bloodstream where they begin the process of breaking down the buildup in the muscles, connective tissue and blood. These enzymes bring nutrition and oxygen-rich blood that can remove the metabolic waste produced by inflammation and excess fibrin.

Serrapeptase has been proven to be the strongest of the proteolytic enzymes, inducing anti-inflammatory, fibrinolytic and anti-edemic (prevents swelling and fluid retention) activity in a number of tissues.

Using enzymes to clear your body of fibrin takes time. It takes years to develop webs of fibrin in your tissues — so be patient, log your usage and, over time, notice how much less pain and how much more flexibility you have.

Serrapeptase has demonstrated anti-inflammatory and fibrinolytic activity, and acts rapidly on localized inflammation with no reports of adverse effects.


Bromelain, a proteolytic enzyme extracted from pineapple, has also been found to be effective in reducing inflammation by blocking cytokine production and activity



Buy on EBay