Tuesday, November 7, 2017

Serracor and #Fibromyalgia Facts

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Fibromyalgia 


Before 1976, fibromyalgia was most commonly known as fibrositis, where “itis” implied an inflammatory component. Despite the understanding of inflammatory pathways to pain, clinical research was unable to identify the role of inflammation in fibromyalgia for many years.

Within the last decade, fibromyalgia research has once again been focusing on the possible contribution of inflammation to disease progression, and is finding some new and interesting results.

Clinical studies have produced evidence that fibromyalgia is associated with the immune system’s improper regulation of proinflammatory cytokines that circulate in the bloodstream, contributing to the dysfunction of the central nervous system and pain-related neurotransmitters. Cytokines, depending on their concentration, induce symptoms, such as fatigue, fever, sleep, pain, and muscle pain, all of which develop in fibromyalgia patients.

These findings are uncovering new possibilities in research for fibromyalgia causation, as well as treatment options. Some experimental pain reduction therapies have been examined and shown positive results, correlating with decreased proinflammatory cytokine levels.7 Anticonvulsant drugs, analgesics, opiods and anti-depressants are commonly prescribed to fibromyalgia patients, but tend to carry side effects reflective of the syndrome itself,and many of which lack evidence for effectiveness.

Limited treatment options have led to an increasing use of systemic enzyme therapy as a means to alleviate symptoms and improve quality of life. Certain proteolytic (protein digesting) enzymes have been identified to have extremely beneficial actions when applied to inflammation and pain related to this condition.
 

It has long been known that people with chronic muscle pain or fibromyalgia have more fibrin in their tissues and blood. This fibrin, while initially helpful in the early stages of healing after an injury, can become problematic if the body does not clear itself of the agent after it has done its work.

Fibromyalgia sufferers experience micro-tears in their muscles from the normal activity of daily living — each and every day. But because the average fibromyalgia patient does not achieve and stay in stage 4 delta sleep at rest, growth hormone is not produced in enough quantities to heal these tears, which leads to more fibrin buildup.

For the most part, people with fibromyalgia do not have a strong enzymatic capacity for producing enzymes that break down fibrin. This leads to a buildup of fibrin, which over time catches red blood cells in a web of restriction. This fibrin causes a restriction of blood flow. Red blood cells literally become stuck, disabling them from getting into the capillaries to oxygenate and nourish the muscles where the metabolic waste that causes pain is removed.

The body uses fibrin to help heal itself after an injury. However, if you have poor blood flow and a lack of enzyme activity, fibrin will start to accumulate. If the injured area is slow to heal, fibrin accumulation appears as clumps of scar tissue in the muscles or at a surgical site.

Ultimately, if excess fibrin is present throughout the circulatory system, blood flow is restricted to areas of the body that need it most. Over time, the body compensates for this restriction by increasing its blood pressure. People with excess fibrin suffer from chronic fatigue, slow healing, inflammation and pain, as well as elevated blood pressure.

Proteolytic enzymes taken on an empty stomach break down these proteins into their smallest elements. The enzymes pass through the stomach and intestinal lining, and enter the bloodstream where they begin the process of breaking down the buildup in the muscles, connective tissue and blood. These enzymes bring nutrition and oxygen-rich blood that can remove the metabolic waste produced by inflammation and excess fibrin.

Serrapeptase has been proven to be the strongest of the proteolytic enzymes, inducing anti-inflammatory, fibrinolytic and anti-edemic (prevents swelling and fluid retention) activity in a number of tissues.

Using enzymes to clear your body of fibrin takes time. It takes years to develop webs of fibrin in your tissues — so be patient, log your usage and, over time, notice how much less pain and how much more flexibility you have.

Serrapeptase has demonstrated anti-inflammatory and fibrinolytic activity, and acts rapidly on localized inflammation with no reports of adverse effects.


Bromelain, a proteolytic enzyme extracted from pineapple, has also been found to be effective in reducing inflammation by blocking cytokine production and activity


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Monday, November 6, 2017

The Role of #Fibrin in #Parkinson’s #Disease #Serrscor NK



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Parkinson’s disease is an irreversible nervous system disorder that primarily affects musculo-skeletal movement. While the progressive disorder can cause stiffness and slow movements, signs and symptoms vary greatly among patients.

Common symptoms include: 
Tremors
Slowed Movement
Rigid Muscles
Impaired balance
Impaired Posture 
Speech changes


Parkinson’s disease is caused by the gradual breakdown of brain cells called neurons. The loss of neurons leads to a decrease in dopamine levels, leading to abnormal brain activity. Only 5% of Parkinson’s cases occur before the age of 40, with incidence increasing with age. A majority of cases seem to be sporadic and of unknown origin – however, like most diseases, there is a significant genetic component.

Recent theories about the causes of Parkinson’s disease are based on abnormal fibrin production and deposition in the brain. Also known as the “amyloid hypothesis,” researchers have been looking at the role of abnormal protein aggregation in Parkinson’s and other neurodegenerative disorders, like Alzheimer’s disease. There seems to be a correlation between fibrin deposits and brain tissue degeneration, suggesting that the fibrin contributes to cellular death in these diseases. Similar to Alzheimer’s disease and Huntington’s disease, Parkinson’s disease involves the mis-folding of a specific protein, eventually leading to fibrin deposits.

There are several theories as to the underlying cause of Parkinson’s, from oxidative damage to environmental toxins – but the latest research seeks to understand why the dopamine-containing neurons are affected so greatly. In fact, by the time a Parkinson’s patient presents with symptoms, about 70% of the dopamine-containing neurons have already been lost. Current therapies aim to address dopamine deficiencies, but future therapies seek to address the prevention of neuron degeneration (cellular death of the brain cells).

Although systemic enzymes are implicated for the treatment of Alzheimer’s disease, their use in similar neurodegenerative diseases has yet to be studied. Specifically nattokinase, which has been shown to directly dissolve the aggregated protein found in Alzheimer’s disease, may be an option available to those suffering from Parkinson’s disease.

 Nattokinase is a natural systemic enzyme that helps to decrease fibrin levels in the blood. Nattokinase is also able to reach areas where fibrin has already accumulated – helping to restore blood flow and inhibiting cellular death due to oxidative damage. Perhaps the most promising aspect of systemic enzyme therapy is its ability to be used concurrently with other medications. The only restriction is upon the use of nattokinase, which should be discussed with a doctor prior to adding it to a regimen with prescription blood thinners. However, it should be noted that systemic enzymes do not cause adverse side effects or drug-drug/drug-nutrient interactions.


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Sunday, November 5, 2017

Systemic Enzymes for #Parkinson's #Excellacor





The Common Cause of Parkinson’s
By William Wong ND, PhD, Member World Sports Medicine Hall of Fame

The road to discovery is long and winding. Some of its paths lead to dead ends, some of its paths curve round and round until they lead to truth. The paths trod by those seeking the cause and then a cure for the ills that effect the brain are no different.

In the search for the root cause of Alzheimer's many degenerative changes in the brain of its victims have come to light: protein cross linking short circuiting nerve transmission, aluminum shards in DNA strands, decrease in neurotransmitters, the shrinking of the brain due to loss of its fat content (the brain is 70% cholesterol), but all of these factors are the results of the condition and not its direct cause. It is now known that moderate inflammation, not enough to be called Enchaphilitis, but enough to have been medically detected, is the root cause of the condition. 

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With Parkinson’s patients a part of the brain called the Substancia Nigra dies off. This vital part of the brain makes a substance called Dopamine. It is dopamine that connects the brain to the body. As dopamine levels decrease while the Substancia Nigra dies off, slowly the control the brain exerts over the body diminishes. So essential is dopamine that doctors can tell a “pre morbid” (just before death) condition by monitoring blood levels of dopamine. Three days after the last drop of dopamine is made by the brain the person dies! A link between sub clinical brain inflammation and Parkinson’s has just been found!


Saturday, November 4, 2017

Serracor-NK for #Fibromyalgia - a disorder of chronic widespread pain


 


Fibromyalgia is a disorder of chronic, widespread pain and tenderness (see the image below). It typically presents in young or middle-aged women but can affect patients of either sex and at any age.
Tender points in fibromyalgia.  
Tender points in fibromyalgia.

Signs and symptoms

Fibromyalgia is a syndrome that consists of the following signs and symptoms
  • Persistent (≥3 mo) widespread pain (pain/tenderness on both sides of the body, above and below the waist, including the axial spine [usually the paraspinus, scapular, and trapezius muscles])
  • Stiffness
  • Fatigue; disrupted and unrefreshing sleep
  • Cognitive difficulties
  • Multiple other unexplained symptoms, anxiety and/or depression, and functional impairment of activities of daily living (ADLs)

Diagnosis

Fibromyalgia is a diagnosis of exclusion and patients must be thoroughly evaluated for the presence of other disorders that could be the cause of symptoms before a diagnosis of fibromyalgia is made. The clinical assessment may reveal objective evidence for a discrete or comorbid illness, such as the following:
  • Hypothyroidism
  • Rheumatoid arthritis
  • Systemic lupus erythematosus
  • Polymyalgia rheumatic
  • Other inflammatory or autoimmune disorders
  • Serious cardiac conditions in those with chest pain, dyspnea, and palpitations
  • Social Support Questionnaire
  • Sickness Impact Profile
  • Multidimensional Pain Inventory
See Workup for more detail.

Management

There is no cure for fibromyalgia, but education, lifestyle changes, and proper medications can help the individual to regain control and achieve significant improvement.
Models of pain behavior that interrelate biologic, cognitive, emotional, and behavioral variables form the basis for cognitive-behavioral and operant behavioral approaches to adult pain management. Fibromyalgia in children responds to a combination of psychotherapy, exercise, relaxation techniques, and education. Pharmacotherapy is generally not indicated in children.
Nonpharmacotherapy
  • Diet (eg, promote good nutrition, vitamin supplementation, bone health, weight loss)
  • Stress management
  • Aerobic exercise (eg, low-impact aerobics, walking, water aerobics, stationary bicycle)
  • Sleep therapy (eg, education/instruction on sleep hygiene)
  • Psychologic/behavioral therapy (eg, cognitive-behavioral, operant-behavioral)
Other agents used in fibromyalgia may include the following:
  • Vitamins and minerals
  • Malic acid and magnesium combination
  • Antioxidants
  • Amino acids
  • Herbs and supplements

Friday, November 3, 2017

Floracor 5 ways you are harming your gut bacteria

 

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5 ways you are harming your gut bacteria

Our society has adopted some very unhealthy activities and lifestyles that affect our microbes in a variety of ways. Most are difficult to avoid, but it's critical to spread awareness if we want to protect our microbiome.

A microbiota is an "ecological community of commensal, symbiotic and pathogenic microorganisms" found in and on all multi cellular organisms studied to date from plants to animals. A microbiota includes bacteria, archaea, protists, fungi and viruses. Here are just a few easy ways you can understand how bacteria works.
  

Wine and beer. Alcohol consumption can alter your microbial balance



Sweets. Processed sugars can feed potentially unhealthy bacteria




Poor sleep. This can lead to subtle changes in your microbial makeup






Antibiotics. These kill both good and bad bacteria.



Tap water. Chlorine in tap water kills both good and bad bacteria.



These are just a few of the most common culprits. If any of these apply to you, it may be time to look for a probiotic.


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Thursday, November 2, 2017

#Cystic #Fibrosis Conference #Serracor-NK #NACFC


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What is cystic fibrosis?

Cystic fibrosis (CF) is a genetic condition, which means that it is something you are born with. CF is known to cause your lungs to produce extra-thick, sticky mucus. This mucus builds up and clogs your airways.






What makes healthy lungs "healthy"?

The lungs naturally produce a thin layer of mucus that helps protect the airways from dangerous bacteria that can cause infections.




How are lungs affected by CF different?

For people living with CF, the mucus in the lungs is thicker than normal. This thick mucus can build up in the airways, allowing germs to thrive and become a source of infection and inflammation that can lead to decreased lung function.

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Wednesday, November 1, 2017

Benefits of #Serrapeptase



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In the U.S., Serrapeptase is classified as a dietary supplement. Serrapeptase is used for painful conditions including back pain, osteoarthritis, rheumatoid arthritis, osteoporosis, fibromyalgia, carpel tunnel syndrome, migraine headache, and tension headache.



What foods contain serrapeptase?
While there are no foods that actually contain serrapeptase, doctors who recommend this enzyme suggest that people to stick to a mainly Mediterranean diet. Eat organic meats, fish and poultry. Beans, nuts, fruit and vegetables should also be incorporated. Whole foods and raw foods are the best.
 Serrapeptase is thought to work in three ways:
  1. It may reduce inflammation by thinning the fluids formed from injury, and facilitating the fluid’s drainage. This in turn, also speeds tissue repair.
  2. It may help alleviate pain by inhibiting the release of pain-inducing amines called bradykinin.
  3. It may enhance cardiovascular health by breaking down the protein by-products of blood coagulation called fibrin. Conveniently, Serrapeptase is able to dissolve the fibrin and other dead or damaged tissue without harming living tissue. This could enable the dissolution of atherosclerotic plaques without causing any harm to the inside of the arteries. 

Serrapeptase has been used in Europe and Asia for over 25 years. Because the enzyme digests or dissolves all nonliving tissue, including blood clots, cysts and arterial plaque, it is used to treat a variety of conditions, including sprains and torn ligaments, postoperative swelling, venous thrombosis (clots in the legs), ear, nose and throat infections and atherosclerosis.

Abroad, Serrapeptase is marketed under a variety of names including, DanzenTM, AniflazymTM, and SerraZymeTM. In the United States, it has been used and marketed as Serrapeptase since 1997. A pain-reliever and anti-inflammatory supplement that has anti-clotting activities without the risk of stomach bleeding? Sounds too good to be true? Let’s look at the research.